Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000697020 | SCV000825610 | uncertain significance | Pyridoxine-dependent epilepsy | 2021-08-12 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Ser16Asnfs*42) in the ALDH7A1 gene. However, it is currently unclear if variants that occur in this region of the gene cause disease. The frequency data for this variant in the population databases is considered unreliable, as metrics indicate poor data quality at this position in the ExAC database. This variant has not been reported in the literature in individuals affected with ALDH7A1-related conditions. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV002332462 | SCV002630883 | uncertain significance | Inborn genetic diseases | 2019-07-12 | criteria provided, single submitter | clinical testing | The c.43_46dupACCA variant, located in coding exon 1 of the ALDH7A1 gene, results from a duplication of ACCA at nucleotide position 43, causing a translational frameshift with a predicted alternate stop codon (p.S16Nfs*42). This alteration is expected to result in loss of function by premature protein truncation or nonsense-mediated mRNA decay; however this variant is located upstream of an alternate in-frame translation initiation codon. Since this may result in expression of a different isoform and the biological contribution of each isoform is not well established, the clinical significance of this alteration remains unclear. |
Genome- |
RCV000697020 | SCV004050465 | uncertain significance | Pyridoxine-dependent epilepsy | 2023-04-11 | criteria provided, single submitter | clinical testing |