Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Baylor Genetics | RCV001027886 | SCV004195940 | pathogenic | Retinitis pigmentosa 28 | 2024-02-14 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV003727839 | SCV004536976 | pathogenic | not provided | 2024-12-02 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Lys315*) in the FAM161A gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in FAM161A are known to be pathogenic (PMID: 20705278, 20705279, 24651477). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with retinitis pigmentosa (PMID: 31236346). ClinVar contains an entry for this variant (Variation ID: 828065). For these reasons, this variant has been classified as Pathogenic. |
Fulgent Genetics, |
RCV001027886 | SCV005657107 | pathogenic | Retinitis pigmentosa 28 | 2024-04-03 | criteria provided, single submitter | clinical testing | |
OMIM | RCV001027886 | SCV001190609 | pathogenic | Retinitis pigmentosa 28 | 2020-03-26 | no assertion criteria provided | literature only |