Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002579065 | SCV003490329 | uncertain significance | HNSHA due to aldolase A deficiency | 2023-06-13 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may create or strengthen a splice site. ClinVar contains an entry for this variant (Variation ID: 2175229). This variant has not been reported in the literature in individuals affected with ALDOA-related conditions. This variant is present in population databases (no rsID available, gnomAD 0.003%). This sequence change falls in intron 7 of the ALDOA gene. It does not directly change the encoded amino acid sequence of the ALDOA protein. |
Prevention |
RCV004747208 | SCV005360158 | uncertain significance | ALDOA-related disorder | 2024-07-25 | no assertion criteria provided | clinical testing | The ALDOA c.113-9C>G variant is predicted to interfere with splicing. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.0029% of alleles in individuals of Latino descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |