Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ambry Genetics | RCV000241668 | SCV000320406 | uncertain significance | Cardiovascular phenotype | 2019-06-24 | criteria provided, single submitter | clinical testing | The p.R10222H variant (also known as c.30665G>A), located in coding exon 123 of the TTN gene, results from a G to A substitution at nucleotide position 30665. The arginine at codon 10222 is replaced by histidine, an amino acid with highly similar properties. This amino acid position is highly conserved in available vertebrate species. In addition, this alteration is predicted to be tolerated by in silico analysis. Since supporting evidence is limited at this time, the clinical significance of this variant remains unclear. |
Labcorp Genetics |
RCV000475954 | SCV000542708 | uncertain significance | Dilated cardiomyopathy 1G; Autosomal recessive limb-girdle muscular dystrophy type 2J | 2017-10-24 | criteria provided, single submitter | clinical testing | |
Laboratory for Molecular Medicine, |
RCV000611588 | SCV000712932 | uncertain significance | not specified | 2017-01-20 | criteria provided, single submitter | clinical testing | The p.Arg16719His variant in TTN has not been previously reported in individuals with cardiomyopathy, but has been identified in 3/11432 Latino chromosomes by t he Exome Aggregation Consortium (ExAC, http://exac.broadinstitute.org; dbSNP rs3 71422299). This variant has been reported in ClinVar (Variant ID: 264456). Compu tational prediction tools and conservation analysis suggest that the p.Arg16719H is variant may impact the protein, though this information is not predictive eno ugh to determine pathogenicity. In summary, the clinical significance of the p.A rg16719His variant is uncertain. |
Gene |
RCV000839331 | SCV000981227 | likely benign | not provided | 2019-09-27 | criteria provided, single submitter | clinical testing |