Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000547535 | SCV000643716 | uncertain significance | Dilated cardiomyopathy 1G; Autosomal recessive limb-girdle muscular dystrophy type 2J | 2017-08-26 | criteria provided, single submitter | clinical testing | |
Eurofins Ntd Llc |
RCV000594551 | SCV000702066 | uncertain significance | not provided | 2016-10-20 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002341356 | SCV002644982 | likely benign | Cardiovascular phenotype | 2019-08-20 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
Fulgent Genetics, |
RCV002476171 | SCV002776652 | uncertain significance | Dilated cardiomyopathy 1G; Autosomal recessive limb-girdle muscular dystrophy type 2J; Tibial muscular dystrophy; Myopathy, myofibrillar, 9, with early respiratory failure; Early-onset myopathy with fatal cardiomyopathy; Hypertrophic cardiomyopathy 9 | 2021-12-03 | criteria provided, single submitter | clinical testing | |
Women's Health and Genetics/Laboratory Corporation of America, |
RCV003330778 | SCV004038652 | uncertain significance | not specified | 2023-08-19 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV004735608 | SCV005349313 | uncertain significance | TTN-related disorder | 2024-07-15 | no assertion criteria provided | clinical testing | The TTN c.78914C>G variant is predicted to result in the amino acid substitution p.Ser26305Cys. To our knowledge, this variant has not been reported in the literature. This variant is reported in 0.079% of alleles in individuals of African descent in gnomAD. Although we suspect that this variant may be benign, at this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |