Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Laboratory for Molecular Medicine, |
RCV000037215 | SCV000060872 | likely pathogenic | Cardiomyopathy | 2012-02-13 | criteria provided, single submitter | clinical testing | The Met1? variant (CRYAB) has not been reported in the literature and has not be en previously detected by our laboratory. This variant is predicted to severely affect the synthesis of the CRYAB protein by disrupting the translation initiat ion start codon (ATG). In summary, this variant is likely to be pathogenic. |
Gene |
RCV001787334 | SCV000680849 | pathogenic | not provided | 2021-11-24 | criteria provided, single submitter | clinical testing | Initiation codon variant in a gene for which loss-of-function is a known mechanism of disease; This variant is associated with the following publications: (PMID: 31215171, 33834702) |
Labcorp Genetics |
RCV001358809 | SCV001554661 | pathogenic | Dilated cardiomyopathy 1II | 2023-12-12 | criteria provided, single submitter | clinical testing | This sequence change affects the initiator methionine of the CRYAB mRNA. The next in-frame methionine is located at codon 68. This variant is present in population databases (rs397516686, gnomAD 0.1%), including at least one homozygous and/or hemizygous individual. Disruption of the initiator codon has been observed in individuals with autosomal recessive myofibrillar myopathy (PMID: 31215171, 33834702). ClinVar contains an entry for this variant (Variation ID: 44236). For these reasons, this variant has been classified as Pathogenic. |
Mendelics | RCV002247423 | SCV002518163 | uncertain significance | not specified | 2022-05-04 | criteria provided, single submitter | clinical testing | |
Revvity Omics, |
RCV001787334 | SCV003828666 | uncertain significance | not provided | 2021-03-24 | criteria provided, single submitter | clinical testing |