Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Victorian Clinical Genetics Services, |
RCV002272872 | SCV002557304 | uncertain significance | Intellectual disability, autosomal dominant 39 | 2019-08-28 | criteria provided, single submitter | clinical testing | A heterozygous missense variant was identified, NM_015025.3(MYT1L):c.634G>A in exon 10 of 25 of the MYT1L gene. This substitution is predicted to create a minor amino acid change from alanine to threonine at position 212 of the protein, NP_055840.2(MYT1L):p.(Ala212Thr). The alanine at this position has very high conservation (100 vertebrates, UCSC), but is not situated in a known functional domain. In silico software predicts this variant to be pathogenic (PolyPhen, SIFT, CADD, MutationTaster). The variant is not present in the gnomAD population database. The variant has not previously been reported in clinical cases. Based on information available at the time of curation, this variant has been classified as a VARIANT of UNCERTAIN SIGNIFICANCE (VUS). |
Gene |
RCV003120869 | SCV003798924 | uncertain significance | not provided | 2023-02-03 | criteria provided, single submitter | clinical testing | Not observed at significant frequency in large population cohorts (gnomAD); In silico analysis supports that this missense variant does not alter protein structure/function; Has not been previously published as pathogenic or benign to our knowledge |
Laboratory of Molecular Genetics |
RCV003389079 | SCV004101224 | uncertain significance | Neurodevelopmental disorder | 2023-02-06 | criteria provided, single submitter | clinical testing |