Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ambry Genetics | RCV001266082 | SCV001444254 | pathogenic | Inborn genetic diseases | 2020-09-22 | criteria provided, single submitter | clinical testing | The c.1675C>T (p.R559*) alteration, located in coding exon 17 of the DLG4 gene, results from a C to T substitution at nucleotide position 1675. This changes the amino acid from an arginine (R) to a stop codon at amino acid position 559. This alteration is expected to result in loss of function by premature protein truncation or nonsense-mediated mRNA decay. Based on data from the Genome Aggregation Database (gnomAD), the DLG4 c.1675C>T alteration was not observed, with coverage at this position. Based on the available evidence, this alteration is classified as pathogenic. |
Institute of Human Genetics, |
RCV001799753 | SCV002044359 | pathogenic | Intellectual developmental disorder 62 | 2024-02-19 | criteria provided, single submitter | clinical testing | Criteria applied: PVS1,PS2,PS4_MOD,PM2_SUP |
Tumer Group, |
RCV001799753 | SCV003915504 | pathogenic | Intellectual developmental disorder 62 | 2023-02-28 | criteria provided, single submitter | research | |
Gene |
RCV004720832 | SCV005327366 | pathogenic | not provided | 2024-01-16 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 38135915, 36854414, 33597769) |