Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000732963 | SCV000860965 | uncertain significance | not provided | 2018-04-25 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001050123 | SCV001214216 | uncertain significance | Peroxisome biogenesis disorder 2B | 2022-11-01 | criteria provided, single submitter | clinical testing | This sequence change replaces arginine, which is basic and polar, with glutamine, which is neutral and polar, at codon 480 of the PEX5 protein (p.Arg480Gln). This variant is present in population databases (rs759523235, gnomAD 0.1%). This variant has not been reported in the literature in individuals affected with PEX5-related conditions. ClinVar contains an entry for this variant (Variation ID: 596979). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is not expected to disrupt PEX5 protein function. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Prevention |
RCV004748946 | SCV005360604 | uncertain significance | PEX5-related disorder | 2024-09-25 | no assertion criteria provided | clinical testing | The PEX5 c.1439G>A variant is predicted to result in the amino acid substitution p.Arg480Gln. This variant was reported as a variant of uncertain significance in an individual with inherited retinal dystrophy (Table S12, Diñeiro et al 2020. PubMed ID: 32483926). This variant is reported in 0.099% of alleles in individuals of Latino descent in gnomAD. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |