Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Genomic Research Center, |
RCV000414932 | SCV000746867 | likely pathogenic | Intellectual disability, X-linked 102 | 2017-12-18 | criteria provided, single submitter | clinical testing | |
Gene |
RCV004783781 | SCV005396402 | likely pathogenic | not provided | 2024-05-10 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 26235985, 30734472, 32852922, 37904618, 32135084, 27959697, 30349862, 32714884) |
Baylor Genetics | RCV000414932 | SCV000328816 | pathogenic | Intellectual disability, X-linked 102 | 2016-05-01 | no assertion criteria provided | clinical testing | Our laboratory has reported dual molecular diagnoses in COL7A1 (NM_000094.3:c.6900+4A>G) and DDX3X (NM_001356.3:c.1052G>A) in an individual with microcephaly, delayed motor milestones, delayed speech, hypotonia, hyperreflexia, repetitive behaviors, dysmorphic features, mild conductive hearing loss, mild epidermolysis bullosa (EB) and a history of prematurity and genital anomalies. |
Gene |
RCV000414932 | SCV002098135 | not provided | Intellectual disability, X-linked 102 | no assertion provided | literature only | Observed in the hemizygous state in an affected male & in the heterozygous state in an unaffected female relative [Snijders Blok et al 2015, Wang et al 2018, Lennox et al 2020] |