Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Counsyl | RCV000675015 | SCV000800442 | likely pathogenic | Smith-Lemli-Opitz syndrome | 2018-06-07 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV000675015 | SCV002811643 | likely pathogenic | Smith-Lemli-Opitz syndrome | 2024-04-05 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV004748898 | SCV005348489 | likely pathogenic | DHCR7-related disorder | 2024-05-03 | no assertion criteria provided | clinical testing | The DHCR7 c.861C>A variant is predicted to result in the amino acid substitution p.Asn287Lys. This variant has been reported, in the homozygous or presumed compound heterozygous state, in individuals with clinical and biochemical features consistent with Smith-Lemli-Opitz syndrome (SLOS) (Yu et al. 2000. PubMed ID: 10814720; Lee et al. 2013. PubMed ID: 23918729). This variant is reported in 0.0026% of alleles in individuals of European (Non-Finnish) descent in gnomAD. This variant is interpreted as likely pathogenic. |