Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001008000 | SCV001167732 | likely pathogenic | not provided | 2019-01-29 | criteria provided, single submitter | clinical testing | The R2472X variant in the LRBA gene has not been reported previously as a pathogenic variant nor as a benign variant, to our knowledge. This variant is predicted to cause loss of normal protein function either through protein truncation or nonsense-mediated mRNA decay. The R2472X variant is not observed in large population cohorts (Lek et al., 2016). We interpret R2472X as a likely pathogenic variant. |
Labcorp Genetics |
RCV001860584 | SCV002236902 | pathogenic | Combined immunodeficiency due to LRBA deficiency | 2022-01-08 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 816968). This variant has not been reported in the literature in individuals affected with LRBA-related conditions. This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Arg2472*) in the LRBA gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in LRBA are known to be pathogenic (PMID: 26206937, 26768763). |