Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Invitae | RCV000705218 | SCV000834204 | uncertain significance | Neuronal ceroid lipofuscinosis 7 | 2022-09-01 | criteria provided, single submitter | clinical testing | This sequence change replaces proline, which is neutral and non-polar, with threonine, which is neutral and polar, at codon 356 of the MFSD8 protein (p.Pro356Thr). This variant is present in population databases (rs756204684, gnomAD 0.003%). This missense change has been observed in individual(s) with neuronal ceroid lipofuscinosis type 7 (PMID: 31489614). ClinVar contains an entry for this variant (Variation ID: 581405). Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Tolerated"; PolyPhen-2: "Probably Damaging"; Align-GVGD: "Class C0"). In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Mendelics | RCV000705218 | SCV001136772 | likely pathogenic | Neuronal ceroid lipofuscinosis 7 | 2020-11-08 | criteria provided, single submitter | clinical testing | The substitution p.Pro356Thr is a highly conserved and rare variant (found in heterozygous state in 4 among 251290 alleles in GnomAD v2.0). It was seen in a homozygous state in 1 and in a compound heterozygous state in 2 patients with CLN7. We classify this variant as likely pathogenic |
Natera, |
RCV001825394 | SCV002084767 | uncertain significance | Late-infantile neuronal ceroid lipofuscinosis | 2019-11-11 | no assertion criteria provided | clinical testing |