Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000684990 | SCV000812458 | uncertain significance | Neuronal ceroid lipofuscinosis 7 | 2022-05-17 | criteria provided, single submitter | clinical testing | This sequence change replaces asparagine, which is neutral and polar, with lysine, which is basic and polar, at codon 6 of the MFSD8 protein (p.Asn6Lys). This variant is present in population databases (rs771879274, gnomAD 0.1%). This variant has not been reported in the literature in individuals affected with MFSD8-related conditions. ClinVar contains an entry for this variant (Variation ID: 565428). Algorithms developed to predict the effect of missense changes on protein structure and function (SIFT, PolyPhen-2, Align-GVGD) all suggest that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Genome- |
RCV000684990 | SCV001712368 | uncertain significance | Neuronal ceroid lipofuscinosis 7 | 2021-05-18 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002544709 | SCV003719648 | uncertain significance | Inborn genetic diseases | 2021-12-21 | criteria provided, single submitter | clinical testing | The c.18C>G (p.N6K) alteration is located in exon 2 (coding exon 1) of the MFSD8 gene. This alteration results from a C to G substitution at nucleotide position 18, causing the asparagine (N) at amino acid position 6 to be replaced by a lysine (K). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Natera, |
RCV001830479 | SCV002084816 | uncertain significance | Late-infantile neuronal ceroid lipofuscinosis | 2019-10-28 | no assertion criteria provided | clinical testing |