Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Genomic Diagnostic Laboratory, |
RCV000238882 | SCV000296993 | likely benign | not specified | 2015-11-20 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000911281 | SCV001056343 | benign | Wiskott-Aldrich syndrome 2 | 2024-10-26 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV002518507 | SCV003589013 | uncertain significance | Inborn genetic diseases | 2024-01-09 | criteria provided, single submitter | clinical testing | The c.208G>A (p.G70S) alteration is located in exon 4 (coding exon 3) of the WIPF1 gene. This alteration results from a G to A substitution at nucleotide position 208, causing the glycine (G) at amino acid position 70 to be replaced by a serine (S). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Gene |
RCV004772885 | SCV005384297 | uncertain significance | not provided | 2024-01-04 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant does not alter protein structure/function; Has not been previously published as pathogenic or benign to our knowledge |
Prevention |
RCV004757178 | SCV005352542 | likely benign | WIPF1-related disorder | 2024-09-09 | no assertion criteria provided | clinical testing | This variant is classified as likely benign based on ACMG/AMP sequence variant interpretation guidelines (Richards et al. 2015 PMID: 25741868, with internal and published modifications). |