Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Ce |
RCV001310722 | SCV001500631 | pathogenic | not provided | 2021-01-01 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV003447245 | SCV005836390 | pathogenic | Charcot-Marie-Tooth disease axonal type 2O | 2024-08-22 | criteria provided, single submitter | clinical testing | This sequence change replaces valine, which is neutral and non-polar, with methionine, which is neutral and non-polar, at codon 612 of the DYNC1H1 protein (p.Val612Met). This variant is not present in population databases (gnomAD no frequency). This missense change has been observed in individuals with congenital myopathy and/or spinal muscular atrophy (PMID: 25609763, 31127727). ClinVar contains an entry for this variant (Variation ID: 637518). Invitae Evidence Modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) indicates that this missense variant is expected to disrupt DYNC1H1 protein function with a positive predictive value of 95%. For these reasons, this variant has been classified as Pathogenic. |
Inherited Neuropathy Consortium | RCV000789737 | SCV000929114 | uncertain significance | Neuronopathy, distal hereditary motor, autosomal dominant | no assertion criteria provided | literature only | ||
Inherited Neuropathy Consortium Ii, |
RCV003447245 | SCV004174353 | uncertain significance | Charcot-Marie-Tooth disease axonal type 2O | 2016-01-06 | no assertion criteria provided | literature only |