Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000730345 | SCV000858074 | uncertain significance | not provided | 2017-11-08 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001855636 | SCV002295809 | uncertain significance | Familial aplasia of the vermis; Meckel-Gruber syndrome | 2021-06-29 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Nucleotide substitutions within the consensus splice site are a relatively common cause of aberrant splicing (PMID: 17576681, 9536098). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. ClinVar contains an entry for this variant (Variation ID: 594940). This variant has not been reported in the literature in individuals affected with CC2D2A-related conditions. This variant is not present in population databases (ExAC no frequency). This sequence change affects codon 146 of the CC2D2A mRNA. It is a 'silent' change, meaning that it does not change the encoded amino acid sequence of the CC2D2A protein. This variant also falls at the last nucleotide of exon 7, which is part of the consensus splice site for this exon. |
Fulgent Genetics, |
RCV005036061 | SCV005660494 | uncertain significance | Meckel syndrome, type 6; Joubert syndrome 9; COACH syndrome 2; Retinitis pigmentosa 93 | 2024-05-28 | criteria provided, single submitter | clinical testing |