Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Center for Molecular Medicine, |
RCV001030031 | SCV001190549 | pathogenic | Inflammatory bowel disease 28 | 2019-05-10 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001030031 | SCV001378746 | pathogenic | Inflammatory bowel disease 28 | 2024-02-08 | criteria provided, single submitter | clinical testing | This sequence change affects codon 179 of the IL10RA mRNA. It is a 'silent' change, meaning that it does not change the encoded amino acid sequence of the IL10RA protein. This variant also falls at the last nucleotide of exon 4, which is part of the consensus splice site for this exon. This variant is present in population databases (no rsID available, gnomAD 0.01%). This variant has been observed in individual(s) with Inflammatory bowel disease (PMID: 26822028, 28267044, 29140941). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 830051). Variants that disrupt the consensus splice site are a relatively common cause of aberrant splicing (PMID: 17576681, 9536098). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. For these reasons, this variant has been classified as Pathogenic. |
Beijing Key Laboratry for Genetics of Birth Defects, |
RCV001030031 | SCV001739465 | likely pathogenic | Inflammatory bowel disease 28 | 2020-02-28 | criteria provided, single submitter | clinical testing | |
Diagnostic Genetics, |
RCV001030031 | SCV003837567 | pathogenic | Inflammatory bowel disease 28 | 2023-01-03 | criteria provided, single submitter | clinical testing | |
Aleixo Muise Laboratory, |
RCV001030031 | SCV005088141 | pathogenic | Inflammatory bowel disease 28 | 2024-07-05 | criteria provided, single submitter | research | PVS1;PS1;PM2;PP3;PP4 |