Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV002239845 | SCV002510384 | pathogenic | Charcot-Marie-Tooth disease type 2 | 2023-07-21 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Trp104*) in the AARS gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in AARS are known to be pathogenic (PMID: 25817015, 28493438, 34446925). This variant is present in population databases (no rsID available, gnomAD 0.01%). This variant has not been reported in the literature in individuals affected with AARS-related conditions. ClinVar contains an entry for this variant (Variation ID: 1682320). For these reasons, this variant has been classified as Pathogenic. |
Mendelics | RCV002247706 | SCV002517471 | pathogenic | Charcot-Marie-Tooth disease axonal type 2N | 2022-05-04 | criteria provided, single submitter | clinical testing | |
DASA | RCV002247706 | SCV002600251 | likely pathogenic | Charcot-Marie-Tooth disease axonal type 2N | 2022-11-03 | criteria provided, single submitter | clinical testing | The c.312G>A;p.(Trp104*) variant creates a premature translational stop signal in the AARS1 gene. It is expected to result in an absent or disrupted protein product - PVS1. The variant is present at low allele frequencies population databases (rs1398433261 – gnomAD 0.00006573%; ABraOM no frequency - https://abraom.ib.usp.br/) - PM2_supporting. In summary, the currently available evidence indicates that the variant is likely pathogenic. |