Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001544991 | SCV001764226 | uncertain significance | not provided | 2024-05-06 | criteria provided, single submitter | clinical testing | Has not been previously published as pathogenic or benign to our knowledge; Not located in the triple helical region, where the majority of pathogenic missense variants occur (HGMD); In silico analysis indicates that this missense variant does not alter protein structure/function |
Labcorp Genetics |
RCV001544991 | SCV002249187 | benign | not provided | 2023-11-02 | criteria provided, single submitter | clinical testing | |
Genetics and Molecular Pathology, |
RCV002466684 | SCV002761870 | uncertain significance | Spondyloperipheral dysplasia | 2021-09-23 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV003365428 | SCV004055274 | uncertain significance | Inborn genetic diseases | 2023-08-21 | criteria provided, single submitter | clinical testing | The c.446G>A (p.R149H) alteration is located in exon 7 (coding exon 7) of the COL2A1 gene. This alteration results from a G to A substitution at nucleotide position 446, causing the arginine (R) at amino acid position 149 to be replaced by a histidine (H). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |