Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000018716 | SCV002240261 | pathogenic | Bethlem myopathy 1A | 2020-10-30 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Studies have shown that this variant is associated with the activation of a cryptic splice site in exon 3 (PMID: 11932968). Disruption of this splice site has been observed in individual(s) with Bethlem myopathy (PMID: 11932968, 28831785, 25535305). It has also been observed to segregate with disease in related individuals. ClinVar contains an entry for this variant (Variation ID: 17176). This variant is not present in population databases (ExAC no frequency). This sequence change affects a donor splice site in intron 3 of the COL6A1 gene. RNA analysis indicates that this variant induces altered splicing and likely results in the loss of 22 amino acid residue(s), but is expected to preserve the integrity of the reading-frame. |
Juno Genomics, |
RCV004795926 | SCV005416601 | pathogenic | Bethlem myopathy 1A; Ullrich congenital muscular dystrophy 1A | criteria provided, single submitter | clinical testing | PVS1_Strong+PM2_Supporting+PS4_Supporting+PP1_Strong+PP4 | |
OMIM | RCV000018716 | SCV000038999 | pathogenic | Bethlem myopathy 1A | 2002-04-01 | no assertion criteria provided | literature only |