Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000481104 | SCV000573448 | uncertain significance | not provided | 2023-08-12 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Has not been previously published as pathogenic or benign to our knowledge |
Eurofins Ntd Llc |
RCV000481104 | SCV000707971 | pathogenic | not provided | 2017-04-26 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV001381261 | SCV001579582 | pathogenic | Bethlem myopathy 1A | 2024-06-19 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg521*) in the COL6A2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in COL6A2 are known to be pathogenic (PMID: 19884007, 20976770). This variant is present in population databases (rs773686174, gnomAD 0.007%). This variant has not been reported in the literature in individuals affected with COL6A2-related conditions. ClinVar contains an entry for this variant (Variation ID: 423719). For these reasons, this variant has been classified as Pathogenic. |
Revvity Omics, |
RCV000481104 | SCV002019665 | pathogenic | not provided | 2020-05-26 | criteria provided, single submitter | clinical testing |