Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001298100 | SCV001487144 | likely benign | not provided | 2024-06-04 | criteria provided, single submitter | clinical testing | |
Gene |
RCV001298100 | SCV001812101 | uncertain significance | not provided | 2025-03-04 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; Not located in the triple helical region, where the majority of pathogenic missense variants occur (HGMD; PMID: 25240749); Has not been previously published as pathogenic or benign to our knowledge; This variant is associated with the following publications: (PMID: 25240749) |
Ambry Genetics | RCV004978257 | SCV005557911 | uncertain significance | Inborn genetic diseases | 2024-10-07 | criteria provided, single submitter | clinical testing | The c.5132C>T (p.T1711I) alteration is located in exon 66 (coding exon 66) of the COL11A1 gene. This alteration results from a C to T substitution at nucleotide position 5132, causing the threonine (T) at amino acid position 1711 to be replaced by an isoleucine (I). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |