Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000522499 | SCV000619149 | pathogenic | not provided | 2024-11-06 | criteria provided, single submitter | clinical testing | Not observed at significant frequency in large population cohorts (gnomAD); Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; This variant is associated with the following publications: (PMID: 37455656, 31526516, 33057194, 35982159, 36153650) |
Ambry Genetics | RCV000624466 | SCV000742553 | pathogenic | Inborn genetic diseases | 2017-05-12 | criteria provided, single submitter | clinical testing | |
Mendelics | RCV000987258 | SCV001136508 | pathogenic | Hepatocellular carcinoma | 2019-05-28 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000522499 | SCV003310640 | pathogenic | not provided | 2022-05-15 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Tyr333*) in the CTNNB1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in CTNNB1 are known to be pathogenic (PMID: 23033978, 24614104, 25326669, 26350204, 28575650). This variant is not present in population databases (gnomAD no frequency). This premature translational stop signal has been observed in individual(s) with CTNNB1-related conditions (PMID: 30640974). ClinVar contains an entry for this variant (Variation ID: 450550). For these reasons, this variant has been classified as Pathogenic. |