Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000594637 | SCV000703898 | uncertain significance | not provided | 2016-12-27 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000594637 | SCV001381722 | uncertain significance | not provided | 2024-01-24 | criteria provided, single submitter | clinical testing | This sequence change replaces asparagine, which is neutral and polar, with threonine, which is neutral and polar, at codon 136 of the LRP5 protein (p.Asn136Thr). This variant is present in population databases (rs759063931, gnomAD 0.004%). This variant has not been reported in the literature in individuals affected with LRP5-related conditions. ClinVar contains an entry for this variant (Variation ID: 498738). Advanced modeling of protein sequence and biophysical properties (such as structural, functional, and spatial information, amino acid conservation, physicochemical variation, residue mobility, and thermodynamic stability) performed at Invitae indicates that this missense variant is not expected to disrupt LRP5 protein function with a negative predictive value of 95%. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Fulgent Genetics, |
RCV002491193 | SCV002781455 | uncertain significance | Bone mineral density quantitative trait locus 1; Exudative vitreoretinopathy 4; Exudative vitreoretinopathy 1; Worth disease; Autosomal dominant osteopetrosis 1; Osteoporosis with pseudoglioma; Osteoporosis; Polycystic liver disease 4 with or without kidney cysts | 2021-08-24 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV003409860 | SCV004107360 | uncertain significance | LRP5-related disorder | 2023-05-11 | criteria provided, single submitter | clinical testing | The LRP5 c.407A>C variant is predicted to result in the amino acid substitution p.Asn136Thr. This variant is reported in 0.0047% of alleles in individuals of European (Non-Finnish) descent in gnomAD (http://gnomad.broadinstitute.org/variant/11-68115630-A-C). A different variant affecting the same amino acid (p.Asn136Lys) was reported in an individual with osteoporosis (Table 2, Caetano da Silva. 2021. PubMed ID: 33939331). At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |