Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001210266 | SCV001381745 | pathogenic | Cranium bifidum occultum | 2019-09-11 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. This variant disrupts the p.Pro148 amino acid residue in MSX2. Other variant(s) that disrupt this residue have been determined to be pathogenic (PMID: 23949913, 23918290). This suggests that this residue is clinically significant, and that variants that disrupt this residue are likely to be disease-causing. This variant has been reported to affect MSX2 protein function (PMID: 9256341, 27013732, 18786927). This variant has been observed to segregate with Boston-type craniosynostosis in a family (PMID: 8106171). ClinVar contains an entry for this variant (Variation ID: 16961). This variant is not present in population databases (ExAC no frequency). This sequence change replaces proline with histidine at codon 148 of the MSX2 protein (p.Pro148His). The proline residue is highly conserved and there is a moderate physicochemical difference between proline and histidine. |
OMIM | RCV000018474 | SCV000038756 | pathogenic | Craniosynostosis 2 | 1996-12-01 | no assertion criteria provided | literature only | |
Gene |
RCV000018474 | SCV000055821 | not provided | Craniosynostosis 2 | no assertion provided | literature only |