Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001036277 | SCV001199632 | likely pathogenic | Methylcobalamin deficiency type cblE | 2024-11-04 | criteria provided, single submitter | clinical testing | This sequence change affects a donor splice site in intron 13 of the MTRR gene. It is expected to disrupt RNA splicing. Variants that disrupt the donor or acceptor splice site typically lead to a loss of protein function (PMID: 16199547), and loss-of-function variants in MTRR are known to be pathogenic (PMID: 15714522). This variant is present in population databases (rs778738842, gnomAD 0.01%). Disruption of this splice site has been observed in individual(s) with clinical features of MTRR-related conditions (PMID: 30041674). ClinVar contains an entry for this variant (Variation ID: 835400). Algorithms developed to predict the effect of sequence changes on RNA splicing suggest that this variant may disrupt the consensus splice site. In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. |
Baylor Genetics | RCV003461431 | SCV004198755 | pathogenic | Neural tube defects, folate-sensitive | 2024-03-22 | criteria provided, single submitter | clinical testing | |
Fulgent Genetics, |
RCV005036304 | SCV005673151 | likely pathogenic | Methylcobalamin deficiency type cblE; Neural tube defects, folate-sensitive | 2024-06-17 | criteria provided, single submitter | clinical testing | |
Natera, |
RCV001036277 | SCV001458156 | likely pathogenic | Methylcobalamin deficiency type cblE | 2020-09-16 | no assertion criteria provided | clinical testing |