Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001754386 | SCV001987500 | uncertain significance | not provided | 2019-05-24 | criteria provided, single submitter | clinical testing | Not observed at a significant frequency in large population cohorts (Lek et al., 2016); In silico analysis, which includes protein predictors and evolutionary conservation, supports a deleterious effect; Has not been previously published as pathogenic or benign to our knowledge |
Ai |
RCV001754386 | SCV002502945 | uncertain significance | not provided | 2021-11-05 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002538733 | SCV003341336 | uncertain significance | Hypertrophic cardiomyopathy 14 | 2022-04-10 | criteria provided, single submitter | clinical testing | In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. Algorithms developed to predict the effect of missense changes on protein structure and function are either unavailable or do not agree on the potential impact of this missense change (SIFT: "Tolerated"; PolyPhen-2: "Probably Damaging"; Align-GVGD: "Class C0"). ClinVar contains an entry for this variant (Variation ID: 1302497). This variant has not been reported in the literature in individuals affected with MYH6-related conditions. This variant is present in population databases (rs766943407, gnomAD 0.01%). This sequence change replaces arginine, which is basic and polar, with glycine, which is neutral and non-polar, at codon 1449 of the MYH6 protein (p.Arg1449Gly). |
Ambry Genetics | RCV005382168 | SCV006034028 | uncertain significance | Cardiovascular phenotype | 2025-02-06 | criteria provided, single submitter | clinical testing | The p.R1449G variant (also known as c.4345A>G), located in coding exon 28 of the MYH6 gene, results from an A to G substitution at nucleotide position 4345. The arginine at codon 1449 is replaced by glycine, an amino acid with dissimilar properties. This amino acid position is conserved. In addition, the in silico prediction for this alteration is inconclusive. Based on the available evidence, the clinical significance of this variant remains unclear. |