Total submissions: 6
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Women's Health and Genetics/Laboratory Corporation of America, |
RCV000037506 | SCV000052974 | benign | not specified | 2019-12-14 | criteria provided, single submitter | clinical testing | |
Laboratory for Molecular Medicine, |
RCV000037506 | SCV000061164 | likely benign | not specified | 2015-10-30 | criteria provided, single submitter | clinical testing | p.Ser1467Ser in exon 31 of MYH6: This variant is not expected to have clinical s ignificance because it does not alter an amino acid residue and is not located w ithin the splice consensus sequence. It has been identified 0.8% (79/10234) of A frican chromosomes, including 1 homozygote by the Exome Aggregation Consortium ( ExAC, http://exac.broadinstitute.org; dbSNP rs150081280). |
Ambry Genetics | RCV000249957 | SCV000319589 | likely benign | Cardiovascular phenotype | 2023-10-13 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |
Gene |
RCV001703426 | SCV000531898 | likely benign | not provided | 2021-06-21 | criteria provided, single submitter | clinical testing | This variant is associated with the following publications: (PMID: 24503780) |
Invitae | RCV000475333 | SCV000557890 | likely benign | Hypertrophic cardiomyopathy 14 | 2024-01-19 | criteria provided, single submitter | clinical testing | |
Ce |
RCV001703426 | SCV004184341 | likely benign | not provided | 2023-11-01 | criteria provided, single submitter | clinical testing | MYH6: BP4, BP7 |