Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001576446 | SCV001803641 | uncertain significance | not provided | 2024-06-11 | criteria provided, single submitter | clinical testing | In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; This variant is associated with the following publications: (PMID: 30942463, 27527345) |
Labcorp Genetics |
RCV001576446 | SCV002298511 | likely benign | not provided | 2025-01-01 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004039446 | SCV004945376 | uncertain significance | Inborn genetic diseases | 2023-11-22 | criteria provided, single submitter | clinical testing | The c.3676C>T (p.R1226W) alteration is located in exon 28 (coding exon 27) of the MYH9 gene. This alteration results from a C to T substitution at nucleotide position 3676, causing the arginine (R) at amino acid position 1226 to be replaced by a tryptophan (W). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |
Prevention |
RCV004733365 | SCV005356507 | uncertain significance | MYH9-related disorder | 2024-04-15 | no assertion criteria provided | clinical testing | The MYH9 c.3676C>T variant is predicted to result in the amino acid substitution p.Arg1226Trp. This variant was reported in an individual with cleft lip (Peng et al. 2016. PubMed ID: 27527345). This variant is reported in 0.040% of alleles in individuals of East Asian descent in gnomAD. Although we suspect this variant may be benign due to the relatively high allele frequency in the general population, at this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |