Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
3billion | RCV002251084 | SCV002521477 | likely pathogenic | Hereditary insensitivity to pain with anhidrosis | 2022-05-22 | criteria provided, single submitter | clinical testing | The variant is not observed in the gnomAD v2.1.1 dataset. Frameshift: predicted to result in a loss or disruption of normal protein function through nonsense-mediated decay (NMD) or protein truncation. Multiple pathogenic variants are reported downstream of the variant. Therefore, this variant is classified as likely pathogenic according to the recommendation of ACMG/AMP guideline. |
Labcorp Genetics |
RCV002251084 | SCV004321732 | pathogenic | Hereditary insensitivity to pain with anhidrosis | 2023-09-08 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (Splice site) in the NTRK1 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in NTRK1 are known to be pathogenic (PMID: 10982191). This variant is not present in population databases (gnomAD no frequency). This variant has not been reported in the literature in individuals affected with NTRK1-related conditions. ClinVar contains an entry for this variant (Variation ID: 1687402). For these reasons, this variant has been classified as Pathogenic. |