Total submissions: 1
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Human Genome Sequencing Center Clinical Lab, |
RCV001537873 | SCV001754775 | likely pathogenic | Arrhythmogenic right ventricular dysplasia 10 | 2019-10-31 | criteria provided, single submitter | clinical testing | The c.565_566insC (p.Glu189AlafsTer44) variant in the PRKAR1A gene is predicted to introduce a premature translation termination codon. It is predicted to cause loss of normal protein function either through abnormal, prematurely truncated protein, or by absence of protein product due to nonsense-mediated mRNA decay. This variant was not observed in the general population (gnomAD database). Loss-of-function variants in PRKAR1A have been identified in multiple individual with Carney complex (PMID: 11115848, 10974026). For these reasons, this variant has been classified as Likely Pathogenic. |