Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Molecular Diagnostics Lab, |
RCV000207500 | SCV000263048 | likely pathogenic | not provided | 2015-07-21 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV002515536 | SCV003442955 | likely pathogenic | RASopathy | 2022-09-02 | criteria provided, single submitter | clinical testing | In summary, the currently available evidence indicates that the variant is pathogenic, but additional data are needed to prove that conclusively. Therefore, this variant has been classified as Likely Pathogenic. This variant, c.175_177del, results in the deletion of 1 amino acid(s) of the MAP2K1 protein (p.Lys59del), but otherwise preserves the integrity of the reading frame. This variant is not present in population databases (gnomAD no frequency). This variant has been observed in individual(s) with cardio-facio-cutaneous syndrome (PMID: 17551924). In at least one individual the variant was observed to be de novo. ClinVar contains an entry for this variant (Variation ID: 222074). Algorithms developed to predict the effect of variants on protein structure and function are not available or were not evaluated for this variant. Experimental studies have shown that this variant affects MAP2K1 function (PMID: 29753091). |
Gene |
RCV000207500 | SCV005376565 | likely pathogenic | not provided | 2024-04-11 | criteria provided, single submitter | clinical testing | In-frame deletion of one amino acid in a non-repeat region; In silico analysis supports a deleterious effect on protein structure/function; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 29753091, 17551924) |
OMIM | RCV002277569 | SCV001244680 | pathogenic | Cardiofaciocutaneous syndrome 3 | 2007-07-01 | no assertion criteria provided | literature only |