Total submissions: 4
| Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
|---|---|---|---|---|---|---|---|---|
| Laboratory for Molecular Medicine, |
RCV000606201 | SCV000731911 | likely benign | not specified | 2017-08-23 | criteria provided, single submitter | clinical testing | p.Arg173Arg in exon 4 of PTPN11: This variant is not expected to have clinical s ignificance because it does not alter an amino acid residue and is not located w ithin the splice consensus sequence. It has been identified in 0.03% (5/16512) o f South Asian chromosomes by the Exome Aggregation Consortium (ExAC, http://exac .broadinstitute.org; dbSNP rs767425313). |
| Labcorp Genetics |
RCV001466747 | SCV001670756 | likely benign | RASopathy | 2024-07-02 | criteria provided, single submitter | clinical testing | |
| Fulgent Genetics, |
RCV002498983 | SCV002808109 | likely benign | Noonan syndrome 1; Juvenile myelomonocytic leukemia; Metachondromatosis; LEOPARD syndrome 1 | 2021-12-14 | criteria provided, single submitter | clinical testing | |
| Ambry Genetics | RCV005260249 | SCV005922104 | likely benign | Cardiovascular phenotype | 2025-02-16 | criteria provided, single submitter | clinical testing | This alteration is classified as likely benign based on a combination of the following: seen in unaffected individuals, population frequency, intact protein function, lack of segregation with disease, co-occurrence, RNA analysis, in silico models, amino acid conservation, lack of disease association in case-control studies, and/or the mechanism of disease or impacted region is inconsistent with a known cause of pathogenicity. |