Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000436760 | SCV000520249 | likely benign | not provided | 2016-12-02 | criteria provided, single submitter | clinical testing | This variant is considered likely benign or benign based on one or more of the following criteria: it is a conservative change, it occurs at a poorly conserved position in the protein, it is predicted to be benign by multiple in silico algorithms, and/or has population frequency not consistent with disease. |
Labcorp Genetics |
RCV002062394 | SCV002334952 | likely benign | RASopathy | 2023-09-01 | criteria provided, single submitter | clinical testing | |
Ambry Genetics | RCV004984870 | SCV005485526 | uncertain significance | Cardiovascular phenotype | 2024-11-18 | criteria provided, single submitter | clinical testing | The c.873A>G variant (also known as p.S291S), located in coding exon 8 of the RAF1 gene, results from an A to G substitution at nucleotide position 873. This nucleotide substitution does not change the serine at codon 291. This variant has been reported in individual(s) in a neuro-cardio-facio-cutaneous syndrome cohort, but clinical details were limited (Justino A et al. Eur J Hum Genet, 2015 Mar;23:347-53). This nucleotide position is not well conserved in available vertebrate species. In silico splice site analysis predicts that this alteration may result in the creation or strengthening of a novel splice acceptor site. Based on the available evidence, the clinical significance of this variant remains unclear. |