Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000198266 | SCV000252329 | likely pathogenic | not provided | 2021-01-28 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is a known mechanism of disease; Not observed at a significant frequency in large population cohorts (Lek et al., 2016); Has not been previously published as pathogenic or benign to our knowledge |
Labcorp Genetics |
RCV000697812 | SCV000826443 | pathogenic | Hereditary spastic paraplegia 7 | 2022-10-02 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Lys559*) in the SPG7 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in SPG7 are known to be pathogenic (PMID: 21623769, 22964162). This variant is present in population databases (rs372981030, gnomAD 0.004%). This premature translational stop signal has been observed in individual(s) with clinical features of SPG7-related conditions (Invitae). ClinVar contains an entry for this variant (Variation ID: 215215). For these reasons, this variant has been classified as Pathogenic. |
Ce |
RCV000198266 | SCV001246898 | pathogenic | not provided | 2019-02-01 | criteria provided, single submitter | clinical testing | |
Genome Diagnostics Laboratory, |
RCV001847888 | SCV002105852 | pathogenic | Hereditary spastic paraplegia | 2017-05-12 | criteria provided, single submitter | clinical testing | |
Genome |
RCV000697812 | SCV004037564 | not provided | Hereditary spastic paraplegia 7 | no assertion provided | phenotyping only | Variant classified as Pathogenic and reported on 01-08-2018 by Invitae. GenomeConnect-Invitae Patient Insights Network assertions are reported exactly as they appear on the patient-provided report from the testing laboratory. Registry team members make no attempt to reinterpret the clinical significance of the variant. Phenotypic details are available under supporting information. |