Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Rady Children's Institute for Genomic Medicine, |
RCV001265552 | SCV001443706 | likely pathogenic | SPTA1-related disorder | criteria provided, single submitter | clinical testing | This variant affects the canonical splice donor site of intron 17 of 51 and is therefore predicted to interfere with splicing and result in loss of normal protein function. This variant has not been previously reported or functionally characterized in the literature to our knowledge. It is absent from the ExAC and gnomAD population databases and thus is presumed to be rare. Multiple splice prediction tools suggest this variant is likely to interfere with normal splicing. Based on the available evidence, the c.2464+1G>A variant is classified as Likely Pathogenic. | |
Mayo Clinic Laboratories, |
RCV001508909 | SCV001715345 | likely pathogenic | not provided | 2020-09-10 | criteria provided, single submitter | clinical testing | PVS1, PM2 |
NIHR Bioresource Rare Diseases, |
RCV001003796 | SCV001162240 | likely pathogenic | Anemia | no assertion criteria provided | research |