Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000686273 | SCV000813784 | likely benign | Kabuki syndrome | 2023-07-07 | criteria provided, single submitter | clinical testing | |
Prevention |
RCV004527738 | SCV004106693 | uncertain significance | KMT2D-related disorder | 2023-06-11 | criteria provided, single submitter | clinical testing | The KMT2D c.7649C>A variant is predicted to result in the amino acid substitution p.Pro2550His. To our knowledge, this variant has not been reported in the literature or in a large population database (http://gnomad.broadinstitute.org), indicating this variant is rare. At this time, the clinical significance of this variant is uncertain due to the absence of conclusive functional and genetic evidence. |
Ambry Genetics | RCV004639320 | SCV005133522 | uncertain significance | Inborn genetic diseases | 2024-05-29 | criteria provided, single submitter | clinical testing | The c.7649C>A (p.P2550H) alteration is located in exon 31 (coding exon 31) of the KMT2D gene. This alteration results from a C to A substitution at nucleotide position 7649, causing the proline (P) at amino acid position 2550 to be replaced by a histidine (H). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |