Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
New York Genome Center | RCV001837253 | SCV002097765 | uncertain significance | Intellectual developmental disorder with gastrointestinal difficulties and high pain threshold | 2021-01-29 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV003698879 | SCV004468769 | uncertain significance | not provided | 2023-12-18 | criteria provided, single submitter | clinical testing | This sequence change replaces serine, which is neutral and polar, with arginine, which is basic and polar, at codon 89 of the PPM1D protein (p.Ser89Arg). This variant is present in population databases (no rsID available, gnomAD no frequency). This variant has not been reported in the literature in individuals affected with PPM1D-related conditions. ClinVar contains an entry for this variant (Variation ID: 1341773). An algorithm developed to predict the effect of missense changes on protein structure and function (PolyPhen-2) suggests that this variant is likely to be tolerated. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. |
Ambry Genetics | RCV004041021 | SCV005008400 | uncertain significance | Inborn genetic diseases | 2024-01-12 | criteria provided, single submitter | clinical testing | The c.267C>G (p.S89R) alteration is located in exon 1 (coding exon 1) of the PPM1D gene. This alteration results from a C to G substitution at nucleotide position 267, causing the serine (S) at amino acid position 89 to be replaced by an arginine (R). Based on insufficient or conflicting evidence, the clinical significance of this alteration remains unclear. |