Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Prevention |
RCV004548052 | SCV004112095 | pathogenic | CDK13-related disorder | 2023-05-18 | criteria provided, single submitter | clinical testing | The CDK13 c.2201A>G variant is predicted to result in the amino acid substitution p.Lys734Arg. This variant has been reported as a de novo finding in individuals with CDK13-related autosomal dominant congenital heart defects, dysmorphic facial features, and intellectual developmental disorder (Lelieveld et al. 2017. PubMed ID: 28867141; 2017. PubMed ID: 28135719; Hamilton et al. 2017. PubMed ID: 29021403; Turner et al. 2019. PubMed ID: 31785789). Additionally, several other missense changes at this same amino acid position have also been reported as de novo and causative (p.Lys734Glu, p.Lys734Thr; Bostwick et al. 2017. PubMed ID: 28807008; https://www.ncbi.nlm.nih.gov/clinvar/variation/1164023/). This variant has not been reported in a large population database (http://gnomad.broadinstitute.org), indicating this variant is rare. This variant is interpreted as pathogenic. |
Gene |
RCV001200021 | SCV001370885 | not provided | Congenital heart defects, dysmorphic facial features, and intellectual developmental disorder | no assertion provided | literature only |