Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Pathology and Clinical Laboratory Medicine, |
RCV001824207 | SCV002073803 | likely pathogenic | Mitochondrial DNA depletion syndrome, encephalomyopathic form with methylmalonic aciduria | criteria provided, single submitter | clinical testing | ||
Prevention |
RCV003394279 | SCV004121169 | pathogenic | SUCLA2-related disorder | 2022-09-19 | criteria provided, single submitter | clinical testing | The SUCLA2 c.1219C>T variant is predicted to result in the amino acid substitution p.Arg407Trp. This variant has been reported in the homozygous state in individuals with Succinate-CoA ligase deficiency (Carrozzo et al. 2016. PubMed ID: 26475597; Garone et al. 2016. PubMed ID: 27651038; Martikainen et al. 2016. PubMed ID: 27111573; Gut et al. 2020. PubMed ID: 33230181; Alkhater et al. 2020. PubMed ID: 33231368). This variant is reported in 0.0033% of alleles in individuals of South Asian descent in gnomAD (http://gnomad.broadinstitute.org/variant/13-48523627-G-A). This variant is interpreted as pathogenic. |
Center for Genomic Medicine, |
RCV001824207 | SCV004804694 | uncertain significance | Mitochondrial DNA depletion syndrome, encephalomyopathic form with methylmalonic aciduria | 2024-03-17 | criteria provided, single submitter | research |