Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Laboratory of Inherited Metabolic Diseases, |
RCV002241666 | SCV002512091 | pathogenic | Acromesomelic dysplasia 1, Maroteaux type | 2022-04-01 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV003770477 | SCV004580563 | pathogenic | Acromesomelic dysplasia 1, Maroteaux type; Tall stature-scoliosis-macrodactyly of the great toes syndrome | 2023-09-29 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg767*) in the NPR2 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in NPR2 are known to be pathogenic (PMID: 15146390, 15572448, 16384845). This variant is present in population databases (no rsID available, gnomAD 0.0009%). This premature translational stop signal has been observed in individual(s) with short stature (PMID: 34006472). ClinVar contains an entry for this variant (Variation ID: 998027). For these reasons, this variant has been classified as Pathogenic. |
Beijing Key Laboratory for Genetic Research of Skeletal Deformity, |
RCV001293708 | SCV001482351 | likely pathogenic | Short stature with nonspecific skeletal abnormalities | 2019-05-31 | no assertion criteria provided | research |