Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Eurofins Ntd Llc |
RCV000727267 | SCV000707100 | pathogenic | not provided | 2017-03-27 | criteria provided, single submitter | clinical testing | |
Labcorp Genetics |
RCV000803254 | SCV000943116 | pathogenic | Duchenne muscular dystrophy | 2022-06-13 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. Studies have shown that this variant results in inclusion of a 58bp pseudoexon between exons 62 and 63 and introduces a premature termination codon (PMID: 32047267). The resulting mRNA is expected to undergo nonsense-mediated decay. ClinVar contains an entry for this variant (Variation ID: 11286). This variant is also known as IVS62-285A>G. This variant has been observed in individuals with Becker muscular dystrophy (PMID: 3393617, 12754707, 20485447, 32047267, 32669210; Invitae). The frequency data for this variant in the population databases is considered unreliable, as metrics indicate insufficient coverage at this position in the gnomAD database. This sequence change falls in intron 62 of the DMD gene. It does not directly change the encoded amino acid sequence of the DMD protein. RNA analysis indicates that this variant induces altered splicing and may result in an absent or disrupted protein product. |
Mendelics | RCV000012038 | SCV002519511 | pathogenic | Becker muscular dystrophy | 2022-05-04 | criteria provided, single submitter | clinical testing | |
OMIM | RCV000012038 | SCV000032272 | pathogenic | Becker muscular dystrophy | 2003-06-01 | no assertion criteria provided | literature only |