Total submissions: 2
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Illumina Laboratory Services, |
RCV001563659 | SCV001786647 | pathogenic | RECQL4-related spectrum disorders | 2021-02-04 | criteria provided, single submitter | clinical testing | The RECQL4 c.1411delC (p.Gln471ArgfsTer87) variant results in a frameshift and is predicted to result in premature termination or absence of the protein. A literature search was performed for the gene, cDNA change, and amino acid change. No publications were found based on this search. The p.Gln471ArgfsTer87 variant is not found in the Genome Aggregation Database in a region of good sequence coverage, so the variant is presumed to be rare. Based on the available evidence and application of the ACMG criteria, the p.Gln471ArgfsTer87 variant is classified as pathogenic for RECQL4-related spectrum disorders. |
Labcorp Genetics |
RCV002570744 | SCV003462568 | pathogenic | Baller-Gerold syndrome | 2022-12-16 | criteria provided, single submitter | clinical testing | For these reasons, this variant has been classified as Pathogenic. ClinVar contains an entry for this variant (Variation ID: 1199231). This variant has not been reported in the literature in individuals affected with RECQL4-related conditions. This variant is not present in population databases (gnomAD no frequency). This sequence change creates a premature translational stop signal (p.Gln471Argfs*87) in the RECQL4 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in RECQL4 are known to be pathogenic (PMID: 12734318, 12952869). |