Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV000227479 | SCV000288253 | pathogenic | Baller-Gerold syndrome | 2023-12-11 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Val1026Alafs*6) in the RECQL4 gene. It is expected to result in an absent or disrupted protein product. Loss-of-function variants in RECQL4 are known to be pathogenic (PMID: 12734318, 12952869). This variant is present in population databases (rs771538008, gnomAD 0.02%). This premature translational stop signal has been observed in individual(s) with Rothmund-Thomson syndrome (PMID: 18716613, 27247962). In at least one individual the data is consistent with being in trans (on the opposite chromosome) from a pathogenic variant. ClinVar contains an entry for this variant (Variation ID: 239754). For these reasons, this variant has been classified as Pathogenic. |
Gene |
RCV000599050 | SCV000710065 | pathogenic | not provided | 2022-07-28 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is a known mechanism of disease; This variant is associated with the following publications: (PMID: 29625052, 31589614, 18716613, 12734318, 27247962) |
Centre for Mendelian Genomics, |
RCV001199106 | SCV001370101 | likely pathogenic | Rapadilino syndrome | 2016-01-01 | criteria provided, single submitter | clinical testing | This variant was classified as: Likely pathogenic. The following ACMG criteria were applied in classifying this variant: PM2,PP5,PP3,PM3. |
Institute of Immunology and Genetics Kaiserslautern | RCV004771469 | SCV005382187 | pathogenic | Rothmund-Thomson syndrome type 2 | 2023-12-18 | criteria provided, single submitter | clinical testing | ACMG Criteria: PM2, PVS1, PM3, PP5; Variant was found in a heterozygous state |