Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV003228005 | SCV003924424 | pathogenic | not provided | 2023-05-11 | criteria provided, single submitter | clinical testing | Not observed at significant frequency in large population cohorts (gnomAD); In silico analysis supports that this missense variant has a deleterious effect on protein structure/function; This variant is associated with the following publications: (PMID: 29610475, 33818783) |
Prevention |
RCV003407833 | SCV004111108 | likely pathogenic | PIK3R2-related disorder | 2022-12-19 | criteria provided, single submitter | clinical testing | The PIK3R2 c.1669G>T variant is predicted to result in the amino acid substitution p.Asp557Tyr. This variant was reported to have occurred de novo in an individual with epilepsy, macrocephaly, and ventriculomegaly (Epilepsy Phenome/Genome Project, Epi4K Consortium 2021. PubMed ID: 33818783). This variant has not been reported in a large population database (http://gnomad.broadinstitute.org), indicating this variant is rare. A different nucleotide substitution affecting the same amino acid (p.Asp557His) has been reported de novo in an individual with megalencephaly-polymicrogyria-polydactyly-hydrocephalus syndrome (Terrone et al. 2016. PubMed ID: 26860062). The c.1669G>T (p.Asp557Tyr) variant is interpreted as likely pathogenic. |
Yale Center for Mendelian Genomics, |
RCV001849843 | SCV002106992 | likely pathogenic | Seizure | 2021-04-05 | no assertion criteria provided | literature only |