Total submissions: 5
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Institute of Human Genetics, |
RCV000014886 | SCV002026272 | pathogenic | Rett syndrome, congenital variant | 2018-01-20 | criteria provided, single submitter | clinical testing | |
Gene |
RCV003441718 | SCV004168958 | pathogenic | not provided | 2023-05-02 | criteria provided, single submitter | clinical testing | Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 19564653) |
Centre for Population Genomics, |
RCV004558253 | SCV005046881 | pathogenic | FOXG1 disorder | 2024-05-27 | criteria provided, single submitter | curation | This variant has been collected from RettBASE and curated to current modified ACMG/AMP criteria. Based on the classification scheme defined by the ClinGen Rett/Angelman-like Expert Panel for Rett/AS-like Disorders Specifications to the ACMG/AMP Variant Interpretation Guidelines VCEP 3.0, this variant is classified as pathogenic. At least the following criteria are met: Predicted to result in loss of function, and LOF is a known mechanism of disease (PVS1). This variant has been identified as a de novo occurrence in an individual with FOXG1 disorder without confirmation of paternity and maternity (PM6, PMID: 19564653). This variant is absent from gnomAD v4.1 (PM2_Supporting). |
OMIM | RCV000014886 | SCV000035141 | pathogenic | Rett syndrome, congenital variant | 2010-01-01 | no assertion criteria provided | literature only | |
Rett |
RCV000014886 | SCV000222379 | pathogenic | Rett syndrome, congenital variant | 2010-07-13 | no assertion criteria provided | curation |