Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV000187457 | SCV000241050 | pathogenic | not provided | 2019-05-28 | criteria provided, single submitter | clinical testing | Previously reported in a patient with myoclonic seizures, absent speech, inability to walk, and no functional hand use (Seltzer et al., 2014); Nonsense variant predicted to result in protein truncation in a gene for which loss-of-function is a known mechanism of disease; Not observed in large population cohorts (Lek et al., 2016); This variant is associated with the following publications: (PMID: 30533527, 24836831, 27640358) |
Institute of Human Genetics, |
RCV001781546 | SCV002026248 | pathogenic | Rett syndrome, congenital variant | 2018-01-20 | criteria provided, single submitter | clinical testing | |
Centre for Population Genomics, |
RCV004558436 | SCV005046857 | pathogenic | FOXG1 disorder | 2024-05-10 | criteria provided, single submitter | curation | This variant has been collected from RettBASE and curated to current modified ACMG/AMP criteria. Based on the classification scheme defined by the ClinGen Rett/Angelman-like Expert Panel for Rett/AS-like Disorders Specifications to the ACMG/AMP Variant Interpretation Guidelines VCEP 3.0, this variant is classified as pathogenic. At least the following criteria are met: Predicted to result in loss of function, and LOF is a known mechanism of disease (PVS1). This variant is absent from gnomAD v4 (PM2_Supporting). Has been observed in at least 2 individuals with phenotypes consistent with FOXG1 disorder (PS4_Supporting). PMID:24836831 PMID:28661489 Variation ID: 205486 |