Total submissions: 3
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Labcorp Genetics |
RCV001873730 | SCV002162725 | uncertain significance | Aortic valve disease 2 | 2022-05-13 | criteria provided, single submitter | clinical testing | ClinVar contains an entry for this variant (Variation ID: 1174564). In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. This premature translational stop signal has been observed in individual(s) with craniosynostosis (PMID: 27606499). This sequence change creates a premature translational stop signal (p.Gln78Glyfs*41) in the SMAD6 gene. It is expected to result in an absent or disrupted protein product. However, the current clinical and genetic evidence is not sufficient to establish whether loss-of-function variants in SMAD6 cause disease. This variant is not present in population databases (gnomAD no frequency). |
Gene |
RCV003442889 | SCV004168098 | likely pathogenic | not provided | 2023-04-14 | criteria provided, single submitter | clinical testing | Frameshift variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss of function is a known mechanism of disease; Not observed at significant frequency in large population cohorts (gnomAD); This variant is associated with the following publications: (PMID: 34953066, 27606499, 34208845) |
The Laboratory of Genetics and Metabolism, |
RCV001799786 | SCV001739409 | pathogenic | Radioulnar synostosis | 2020-06-20 | no assertion criteria provided | research |