Total submissions: 4
Submitter | RCV | SCV | Clinical significance | Condition | Last evaluated | Review status | Method | Comment |
---|---|---|---|---|---|---|---|---|
Gene |
RCV001770724 | SCV001992751 | uncertain significance | not provided | 2019-06-27 | criteria provided, single submitter | clinical testing | Has not been previously published as pathogenic or benign to our knowledge; Not observed in large population cohorts (Lek et al., 2016); Nonsense variant predicted to result in protein truncation or nonsense mediated decay in a gene for which loss-of-function is not a known mechanism of disease |
Labcorp Genetics |
RCV002032855 | SCV002155971 | uncertain significance | RASopathy | 2021-10-11 | criteria provided, single submitter | clinical testing | This sequence change creates a premature translational stop signal (p.Arg786*) in the SOS1 gene. It is expected to result in an absent or disrupted protein product. However, the current clinical and genetic evidence is not sufficient to establish whether loss-of-function variants in SOS1 cause disease. In summary, the available evidence is currently insufficient to determine the role of this variant in disease. Therefore, it has been classified as a Variant of Uncertain Significance. This variant has not been reported in the literature in individuals affected with SOS1-related conditions. This variant is not present in population databases (ExAC no frequency). |
Genome- |
RCV002468299 | SCV002763310 | uncertain significance | Noonan syndrome 4 | criteria provided, single submitter | clinical testing | ||
Genome- |
RCV002468298 | SCV002763311 | uncertain significance | Fibromatosis, gingival, 1 | criteria provided, single submitter | clinical testing |